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As previously described under the subsection of IGF-1R, the authors investigated the let-7 function in glucose metabolism using inducible let-7 transgenic mice model and found that let-7 overexpression induced glucose intolerance by targeting multiple components of the insulin-PI3K-mTOR signaling pathway including IRS-2, IGF-1R, and INSR
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The treatment is typically performed in a clinical setting by a trained healthcare professional
For individuals aged 60 years and older, the coefficient was 0.101 (95% CI -0.165 to 0.037, p = 0.0019), indicating a significant negative correlation
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